The thing about cures for Retrograde Amnesia is that one would go about the treatment on a case by case basis. Many variables are to be considered in approaching a treatment plan. Treating a 25-year-old alcoholic would be much different than treating an 80-year-old woman in a convalescent home. In the case of the alcoholic, abstaining from alcohol would be part of the treatment plan. Psychotherapy and hypnosis could be effective tools for treating such Amnesia if the nature of the cause is emotional. Having a basket full of different possible treatments is in a practitioner’s best interest in addressing Retrograde Amnesia.
Improper organization and categorization of verbal material
Incorrect way of language formation
Long-term memory loss
Difficulty recollecting non-verbal information
Unplanned travel or wandering from home, or workplace
Temporally graded retrograde amnesia
Systemic consolidation, which describes the processes involved in the retrieval of memory, becomes impaired for a temporary period of time. Traces of anterograde amnesia could be found in this type.
Pure retrograde amnesia
The type is marked by the absence of anterograde amnesia.
Focal retrograde amnesia
Physical abnormalities are usually not observed in this form of RA. This condition is more of a psychological type of memory loss with few traits of anterograde amnesia.
Isolated retrograde amnesia
This is an intense form of RA and affected patients normally have extreme difficulty recalling past information.
Retrograde amnesia can strike even the toughest. Scott Bolzan, who played briefly in the NFL, slipped in the bathroom in 2008 and slammed the back of his head on the floor, wiping out 46 years of memory, ABC News reported.
The scrapbook of Kay Delaney's mind is nearly blank. Delaney, 55, slipped and hit her head at work last year, erasing more than 20 years of memories that include raising her three children, according to several reports.
Delaney, 55, believes she is 34. The last thing she can remember is putting her oldest son and daughter to bed in the early 1990s, according to the Telegraph.
The available data suggest that RA can vary considerably in its severity—from temporally limited RA covering up to a few years to more extensive, ungraded RA involving all of the decades covered by the tests
Another major difficulty is that the study of past memory in amnesic patients relies necessarily on retrospective methods and imperfect tests. For tests of factual knowledge (e.g., public events tests), individuals vary widely in how much they know about the subject matter, independent of the influence of amnesia. For tests of autobiographical knowledge, it is often not possible to verify the accuracy of the recollections, and there are also difficult issues concerning how best to score the content of what is remembered.
Retrograde amnesia in its pure form is very rare, and would involve an injury or disease that affected either the hippocampus, which is an area of the brain responsible for long term memory, or the inferior aspects of the temporal lobe, which plays a role in the memory for both visual and auditory events. Retrograde amnesia is usually accompanied by anterograde amnesia, which is amnesia for events that follow the traumatic episode.
Stedmans medical dictionary (2001) defines retrograde amnesia as amnesia in reference to events that occurred before the trauma or disease that caused the condition (pg. 858). Therefore, retrograde amnesia affects the individuals memory for events that occurred prior to the trauma or disease. For example, frequently, individuals involved in automobile accidents do not remember what happened to cause the accident or even being in the accident.
Retrograde amnesia targets your most recent memories first. The more severe the case, the farther back in time the memory loss extends. This pattern of destroying newer memories before older ones is called Ribot's law. It happens because the neural pathways of newer memories are not as strong as older ones that have been strengthened by years of retrieval. Retrograde amnesia usually follows damage to areas of the brain besides the hippocampus because long-term memories are stored in the synapses of different brain regions. For example, damage to Broca's area, which houses language information, would likely cause language-related memory loss.